Abstract
Cytosolic reductases form C13-OL metabolites of anthracycline antibiotics. We evaluated and compared the activities of these enzymes in a model of daunorubicin-induced cardiomyopathy in rabbits in vivo and in isolated hepatocytes
Cytosolic reductases form C13-OL metabolites of anthracycline antibiotics. We evaluated and compared the activities of these enzymes in a model of daunorubicin-induced cardiomyopathy in rabbits in vivo and in isolated hepatocytes