Incidence of helicobacter pvlori in nasal lymphatic tissues and its possible link to adenoidal vegetation
Abstract
Helicobacter pylori (HP) is well known as the main cause of chronic gastritis, gastroduodenal ulcers and gastric cancer. It plays a role in the development of MALT lymphoma as well as other diseases.
HP is also implicated in the pathogenesis of oropahryngeal squamous cell carcinoma. In children, the nasal lymphatic tissues and adenoids, are possibly the source of this HP infection.
It is believed that this infection follows a similar immune and inflammatory process to the one in the stomach or the lymphatic tissue associated to the mucosal MALT: this can lead to lymphatic hypertrophy and adenoid vegetation (AV). So far, a HP colonisation has not been proved nor dismissed.
Our aim is to confirm whether HP infection is linked to lymphatic hypertrophy in the nasal lymphatic tissues or it is just a concomitent occurence.