Abstract
Circadian rhythms are rhythmically repeating and genetically encoded changes in organisms working on many different levels. They are bound to changes in light/dark cycle and control individual sleep-wake cycle, food intake and other processes.
Their disruption is mainly caused by long-term changes in the duration of sleep, exposition to artificial light after sunset, shift-work schedule and eating habits. Disruption of circadian rythms is considered as a risk factors for increased appetite, weight, blood pressure and decreased glucose tolerance or immune response, resulting in development of cardiovascular or immunological disorders.
From the perspective of pathogenesis of metabolic disorders, individual differences in circadian settings - called chronotypes are a scientifically interesting area for further research. The aim of this work is to study the relationship between circadian rhythms, chronotypes and metabolic disorders, e.g. obesity, diabetes or metabolic syndrome.
Due to the obvious influence of exogenous factors and a high comorbidity of the aforementioned health complications, there is a large potential for the improvement of their treatment as well as prevention.