- The role of PU.1 and GATA-1 transcription factors during normal and leukemogenic hematopoiesis2010 | First Faculty of Medicine
- PU.1 Activation Relieves GATA-1-Mediated Repression of Cebpa and Cbfb during Leukemia Differentiation2009 | First Faculty of Medicine
- Delineating aggressiveness of acute myeloid leukemia in a mouse model carrying mutations of Spi1 (PU.1) and Trp53Publication without faculty affiliation
- 5-Azacitidine in aggressive myelodysplastic syndromes regulates chromatin structure at PU.1 gene and cell differentiation capacity2012 | First Faculty of Medicine
- TRANSCRIPTION FACTOR PU.1 IS A TARGET OF 5-AZACITIDINE DURING DIFFERENTIATION THERAPY OF MYELODYSPLASTIC SYNDROMEPublication without faculty affiliation
- Epigenetic silencing of the oncogenic miR-17-92 cluster during PU.1-directed macrophage differentiation2011 | First Faculty of Medicine
- The role of transcription factors PU.1 and GATA-1 during leukemia differentiationPublication without faculty affiliation
- Oncogenic microRNA-155 and its target PU.1: an integrative gene expression study in six of the most prevalent lymphomas2015 | First Faculty of Medicine
- Sustained PU.1 Levels Balance Cell-Cycle Regulators to Prevent Exhaustion of Adult Hematopoietic Stem Cells2013 | Central Library of Charles University
- New medical technologies help patients with MDS2012 | First Faculty of Medicine
- Regulation of gene expression during normal and leukemia differentiationPublication without faculty affiliation
- PU.1-c-Jun interaction is crucial for PU.1 function in myeloid development2022 | Second Faculty of Medicine
- GATA-1 Inhibits PU.1 Gene via DNA and Histone H3K9 Methylation of Its Distal Enhancer in Erythroleukemia2016 | First Faculty of Medicine
- PU.1 expression is modulated by the balance of functional sense and antisense RNAs regulated by a shared cis-regulatory element2008 | Central Library of Charles University
- Antagonismus transkripčních faktorů GATA-1 a PU.1 reguluje diferenciační kapacitu leukemických buněkPublication without faculty affiliation
- The response of FLi1, EKLF, TP53 (p53), PU.1, MDM2 gene expression in 5q- syndrome to lenalidomide treatmentPublication without faculty affiliation
- Mutual regulatory loop between miR-155 and PU.1 is a candidate pathogenesis factor in CLLPublication without faculty affiliation
- PU.1 váže a aktivuje monocytární a granulocytární geny zatímco váže a neaktivuje lymfoidní geny včetně receptoru pro IL7Publication without faculty affiliation
- Are Fli1, EKLF, PU.1, HDM2 and TP53 factors implicated in refractory anemia and megakaryopoiesis in 5q- syndromePublication without faculty affiliation
- PU.1 Dose-dependently induces granulocyte or macrophage commitment by targeting lineage restricted genes and by regulating transcription factors Egr.2, Nab2, Cebpa and Gfi.1Publication without faculty affiliation
- Role of oncogenic microRNA miR-17-92 and miR 155 in hematopoiesis and leukemiaPublication without faculty affiliation
- BCR-ABL1 mediated miR-150 downregulation through MYC contributed to myeloid differentiation block and drug resistance in chronic myeloid leukemia2018 | First Faculty of Medicine, Central Library of Charles University
- Interconversion between Tumorigenic and Differentiated States in Acute Myeloid Leukemia2019 | First Faculty of Medicine
- MYB transcriptionally regulates the miR-155 host gene in chronic lymphocytic leukemia2011 | First Faculty of Medicine
- Epigenetic Control of SPI1 Gene by CTCF and ISWI ATPase SMARCA52014 | First Faculty of Medicine
- Identification of a myeloid committed progenitor as the cancer-initiating cell in acute promyelocytic leukemia2009 | Central Library of Charles University
- Combined Approach to Leukemic Differentiation Using Transcription Factor PU.1-Enhancing Agents2022 | First Faculty of Medicine
- Transcriptional and epigenetic regulation in the development of myeloid cells: normal and diseased myelopoiesis2014 | First Faculty of Medicine
- Aggressive acute myeloid leukemia in PU.1/p53 double-mutant mice2014 | First Faculty of Medicine
- Loss of ISWI ATPase SMARCA5 (SNF2H) in Acute Myeloid Leukemia Cells Inhibits Proliferation and Chromatid Cohesion2020 | First Faculty of Medicine
